The formins are a bustling family of cellular proteins involved in all sorts of basic biology from development to tissue maintenance to wound healing. The formins are well connected because they can regulate actin polymerization at the fast growing, barbed end of the filament. In cell-cell adhesion, F-actin polymerization is central to cadherin stability but to get a better picture of how or even which formins might be involved, ASCB members Megha Vaman Rao and Ronen Zaidel-Bara in the Mechanobiology Institute at the National University of Singapore (NUS), took a closer look. In a new paper, the NUS researchers identified two, mDia1 and Fmnl3, as major players in binding up epithelial junctions during cell-cell adhesion especially during a dynamic and critical process like wound healing.